Retained placenta in
cattle
Definition:
Failure to expel the foetal membranes within 12 to 24 h after calving.
Delayed placental separation predisposes cows to acute
puerperium metritis and endometritis post partum through:
·
increased bacterial multiplication in the uterus
·
impairment of immune function of endometrial
macrophages and neutrophiles by bacterial endotoxins
Cystic ovarian disease (COD) in cattle
Cystic Ovarian Disease (COD) and follicular
persistency can be described as an Anovulatory condition with Follicle Growth
to Ovulatory or Larger Size of Follicle.
Mastitis in dairy cattle
is the persistent, inflammatory reaction of
the udder tissue.
Mastitis occurs when white blood cells (leukocytes), are
released into the mammary gland, usually in response to an invasion of bacteria of the teat
canal. Milk-secreting
tissue, and various ducts throughout the mammary gland are damaged due to toxins by the bacteria.
Mastitis can also occur as a result of chemical,
mechanical, or thermal injury. The udder sac is hard, tight, and firm.
Subclinical ketosis
is defined as high serum ketone body concentrations
without observed clinical signs.
Subclinical ketosis
causes delayed reproductive functions return to normal after calving, increased
intervals from calving to first and last service, and an increased frequency of
ovarian cyst
Hepatic
lipidosis
is a major
metabolic disorder of many dairy cows in early lactation and is associated with
decreased health status and reproductive performance. In severe cases, milk
production and feed intake are decreased. Fatty liver develops when the
hepatic uptake of lipids exceeds the oxidation and secretion of lipids by the
liver, which usually is preceded by high concentrations of plasma NEFA
mobilized from adipose tissue.


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